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NaDOC exerts its effect altering the transcellular Ca 2+ pathway

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Future research should further elucidate the complex interplay between lipid metabolism and AP, focusing on the molecular mechanisms underlying the relationship between hypertriglyceridemia and necrotizing pancreatitis and exploring novel therapeutic strategies to mitigate the damage caused by lipid metabolic disturbances, ultimately improving clinical outcomes in AP

Activation of ERRs can shift myofiber phenotype toward oxidative, increase capillary density and mitochondrial content, and favor lipid use over carbohydrate under resting and submaximal workloads

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